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How to change font size in quicken 20153/16/2024 ![]() ![]() ![]() Patients with Parkinson’s disease (PD) suffer from progressive impairment of motor function caused by a loss of dopaminergic neurones in the basal ganglia and brainstem 1. Further training of deep brain neurofeedback might provide therapeutic benefits for Parkinson patients by improving symptom control using strategies optimized through neurofeedback. ![]() Moreover, we observed motor improvement when the learnt mental strategies were applied 2 days later without neurofeedback. Lastly, endogenous control over deep brain activity was possible even after removing visual neurofeedback, suggesting that neurofeedback-acquired strategies were retained in the short-term. During a single one-hour training session, the reduction of beta-oscillatory activity became gradually stronger and we observed improved motor performance. Patients volitionally controlled ongoing beta-oscillatory activity by visual neurofeedback within minutes of training. We implemented real-time processing of pathological subthalamic beta oscillations through implanted DBS electrodes to provide deep brain electrical neurofeedback. While pharmacological treatment and deep brain stimulation (DBS) reduce these pathological oscillations concomitantly with improving motor performance, we set out to explore neurofeedback as an endogenous modulatory method. Parkinsonian motor symptoms are linked to pathologically increased beta-oscillations in the basal ganglia. ![]()
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